[1,1'-Biphenyl]-4-sulfonamide, N-(3-methylphenyl)- | 291536-80-6

• 分子结构分类: 有机化合物 - 苯类化合物 - 苯和取代衍生物
• 英文名称: [1,1'-Biphenyl]-4-sulfonamide, N-(3-methylphenyl)-
• 英文别名: N-(3-methylphenyl)-4-phenylbenzenesulfonamide
• CAS: 291536-80-6
• 化学式: C₁₉H₁₇NO₂S
• 分子量: 323.4
• InChiKey: NGUSIXXSBWXPQF-UHFFFAOYSA-N

计算性质

• 辛醇/水分配系数(LogP)：
  4.4
• 重原子数：
  23
• 可旋转键数：
  4
• 环数：
  3.0
• sp3杂化的碳原子比例：
  0.05
• 拓扑面积：
  54.6
• 氢给体数：
  1
• 氢受体数：
  3

文献信息

• Fungicide enhancers effective for treating plants infected with fungal pathogens
  申请人：Board of Regents, The University of Texas System

  公开号：US11129383B2

  公开（公告）日：2021-09-28

  The present invention includes compositions and methods of for treating plants infected with fungal pathogens by contacting an infected plant or plant at risk of infection with a fungicidal composition comprising an fungicide selected from copper compound such as copper octanoate or copper hydroxide, or a triazole fungicide such as myclobutanil, propiconazole, tebuconazole or epoxiconazole, an enhancer selected from apyrase inhibitors, e.g., N-(m-tolyl)-[1, 1′-biphenyl]-4-sulfonamide, S-heptyl 2-oxo-2H-chromene-3-carbothioate, 3-(N-(4-bromophenyl) sulfamoyl)-N-(3-nitrophenyl) benzamide, or (E)-3-methyl-N-(1-(naphthalen-2-yl) ethylidene) benzohydrazide and, optionally, a phytologically-acceptable inert carrier.

  本发明包括用于处理受真菌病原体感染的植物的组合物和方法，方法是将受感染的植物或有感染风险的植物与杀真菌组合物接触，该杀真菌组合物包含选自铜化合物如辛酸铜或氢氧化铜，或三唑类杀菌剂如霉唑醇、丙环唑、戊唑醇或环唑醇的杀真菌剂，选自apyrase抑制剂的增强剂，如N-(间甲苯基)-[1，1′-联苯]-4-磺酰胺、2-氧代-2H-苯并吡喃-3-硫代甲酸 S-庚酯、3-(N-(4-溴苯基)氨基磺酰基)-N-(3-硝基苯基)苯甲酰胺或(E)-3-甲基-N-(1-(萘-2-基)亚乙基)苯甲酰肼，以及植物学上可接受的惰性载体。
• FUNGICIDE ENHANCERS EFFECTIVE FOR TREATING PLANTS INFECTED WITH FUNGAL PATHOGENS
  申请人：Board of Regents, The University of Texas System

  公开号：EP3250033A1

  公开（公告）日：2017-12-06
• Regulation of Stomatal Apertures by Apyrases and Extracellular Nucleotides
  申请人：Roux Stanley J.

  公开号：US20140123342A1

  公开（公告）日：2014-05-01

  The role of extracellular nucleotides and apyrase enzymes in the guard cells that border stomata in regulating stomatal aperture and the plant's resistance to drought and pathogens is disclosed herein. Expression of apyrases APY1 and APY2, in guard cell protoplasts is strongly correlated with cell growth, cell secretory activity and with conditions that favor stomatal opening. Both short-term inhibition of ectoapyrase activity and long-term suppression of APY1 and APY2 transcript levels significantly disrupt normal stomatal behavior in light. Furthermore, two punnoceptor inhibitors in mammals, pyridoxalphosphate-6-azo-phenyl-2′,4′-disulphonic acid (PPADS) and Reactive Blue 2, block ATPS- and ADPβS-induced opening and closing, and also partially block the ability of abscisic acid (ABA) to induce stomatal closure, and light-induced stomatal opening. Treatment of epidermal peels with ATPyS induces increased levels of nitric oxide and reactive oxygen species, and genetically suppressing the synthesis of these agents blocks the effects of nucleotides on stomatal aperture.
• US9371286B2
  申请人：——

  公开号：US9371286B2

  公开（公告）日：2016-06-21
• [EN] REGULATION OF STOMATAL APERTURES BY APYRASES AND EXTRACELLULAR NUCLEOTIDES<br/>[FR] RÉGULATION DES OUVERTURES STOMATIQUES PAR DES APYRASES ET DES NUCLÉOTIDES EXTRACELLULAIRES
  申请人：UNIV TEXAS

  公开号：WO2012167023A2

  公开（公告）日：2012-12-06

  The role of extracellular nucleotides and apyrase enzymes in the guard cells that border stomata in regulating stomatal aperture and the plant's resistance to drought and pathogens is disclosed herein. Expression of apyrases APY1 and APY2, in guard cell protoplasts is strongly correlated with cell growth, cell secretory activity and with conditions that favor stomatal opening. Both short-term inhibition of ectoapyrase activity and long-term suppression of APY1 and APY2 transcript levels significantly disrupt normal stomatal behavior in light. Furthermore, two punnoceptor inhibitors in mammals, pyridoxalphosphate-6-azo-phenyl-2', 4 '-disulphonic acid (PPADS) and Reactive Blue 2, block ATPS- and ADPβS-induced opening and closing, and also partially block the ability of abscisic acid (ABA) to induce stomatal closure, and light-induced stomatal opening. Treatment of epidermal peels with ATPyS induces increased levels of nitric oxide and reactive oxygen species, and genetically suppressing the synthesis of these agents blocks the effects of nucleotides on stomatal aperture.