1,2,3,6,7,8-六氯二苯并-对-二恶英 | 57653-85-7

• 分子结构分类: 有机化合物 - 有机杂环化合物 - 苯并二恶英
• 中文名称: 1,2,3,6,7,8-六氯二苯并-对-二恶英
• 英文名称: 1,2,3,6,7,8-hexaCDD
• 英文别名: 1,2,3,6,7,8-Hexachlorodibenzo-P-dioxin
• CAS: 57653-85-7
• 化学式: C₁₂H₂Cl₆O₂
• 分子量: 390.865
• InChiKey: YCLUIPQDHHPDJJ-UHFFFAOYSA-N

物化性质

• 熔点：
  285°C
• 沸点：
  475.35°C (rough estimate)
• 密度：
  1.8350 (estimate)
• 物理描述：
  1,2,3,6,7,8-hexachlorodibenzo-p-dioxin is a fluffy white solid. (NTP, 1992)
• 蒸汽压力：
  3.6X10-11 mm Hg at 25 °C /extrapolated/
• 稳定性/保质期：
  Extremely stable, even on heating to 700 °C. /Chlorodibenzo-para-dioxins/
• 分解：
  When heated to decomposition it emits toxic fumes of /hydrogen chloride/.
• 保留指数：
  2749

计算性质

• 辛醇/水分配系数(LogP)：
  7.4
• 重原子数：
  20
• 可旋转键数：
  0
• 环数：
  3.0
• sp3杂化的碳原子比例：
  0.0
• 拓扑面积：
  18.5
• 氢给体数：
  0
• 氢受体数：
  2

ADMET

代谢

在大鼠中，二苯并-p-二噁英的主要羟基化作用仅发生在2、3、7或8的位置。

In rats, primary hydroxylation of dibenzo-p-dioxins takes place exclusively at 2, 3, 7, or 8 position.

来源:Hazardous Substances Data Bank (HSDB)

代谢

七种多氯二苯并呋喃异构体的代谢在雌性Sprague Dawley大鼠中进行了研究。在给予适当的多氯二苯并呋喃异构体后2小时开始，通过手术植入的胆管导管收集了为期3至7天的胆汁样本。剂量通过静脉注射或口服灌胃给药。从胆汁中分离出代谢物，并通过GS/MS分析为羟基化化合物。四氯二苯并呋喃异构体经历了快速的生物转化。没有看到开环产物，两个异构体都代谢为羟基化的四氯二苯并呋喃和三氯二苯并呋喃，以及二羟基-三氯二苯并呋喃。五氯二苯并呋喃异构体的代谢物在分布上显示出定量差异；1,2,3,4,8-五氯二苯并呋喃主要代谢为羟基-五氯二苯并呋喃，1,2,3,7,8-五氯二苯并呋喃导致二羟基-五氯二苯并呋喃，而2,3,4,7,8-五氯二苯并呋喃的代谢物多样，并且是由于醚键断裂。1,2,3,6,7,8-六氯二苯并呋喃和1,2,3,4,6,7,8-七氯二苯并呋喃没有导致可辨别的代谢物浓度。得出结论，大鼠肝脏中多氯二苯并呋喃的代谢主要是由于氧化、水解或还原脱氯，醚键断裂是一种相对不重要的代谢机制，氯代模式影响代谢，并且随着每个环中超过两个氯原子的存在，多氯二苯并呋喃的代谢减少。

The metabolism of seven polychlorodibenzofuran isomers were studied in female Sprague Dawley rats. Bile samples were taken from surgically implanted bile duct cannulas over a period of 3 to 7 days, beginning 2 hr after dosing with the appropriate polychlorodibenzofuran isomer. Doses were administered either intravenously or by oral gavage. Metabolites were isolated from bile and analyzed as hydroxylated cmpd by GS/MS. The tetrachlorodibenzofuran isomers underwent rapid biotransformation. No ring opened products were seen, and both isomers were metabolized to hydroxylated tetrachlorodibenzofurans and trichlorodibenzofurans, and dihydroxy-trichlorodibenzofurans. The metabolites of the pentachlorodibenzofuran isomers showed quantitative differences in distribution; 1,2,3,4,8-pentachlorodibenzofuran metabolized primarily to hydroxy-pentachlorodibenzofurans, 1,2,3,7,8-pentachlorodibenzofuran resulted in dihydroxy-pentachlorodibenzofurans, and 2,3,4,7,8-pentachlorodibenzofuran metabolites were varied and resulted from ether bond cleavage. Neither 1,2,3,6,7,8-hexachlorodibenzofuran nor 1,2,3,4,6,7,8-heptachlorodibenzofuran resulted in discernible metabolite concn. It was concluded that polychlorodibenzofuran metabolism in rat liver is primarily due to oxidation, or hydrolytic or reductive dechlorination, that ether bond cleavage is a relatively unimportant metabolic mechanism, that chlorine substitution patterns affect metabolism, and that metabolism of polychlorodibenzofurans decreases with more than two chlorines in each ring.

来源:Hazardous Substances Data Bank (HSDB)

代谢

CDDs通过口服、吸入和皮肤接触途径被吸收。CDDs通过血清脂质和脂蛋白在血浆中携带，主要分布到肝脏和脂肪组织。CDDs通过微粒体单加氧酶系统非常缓慢地被代谢为极性代谢物，这些代谢物可以与葡萄糖醛酸和谷胱甘肽结合。它们可能通过诱导I相和II相酶来增加自己的代谢速率。CDDs的主要排泄途径是胆汁和粪便，尽管也有少量通过尿液和哺乳排出。(L177)

CDDs are absorbed through oral, inhalation, and dermal routes of exposure. CDDs are carried in the plasma by serum lipids and lipoproteins, distributing mainly to the liver and adipose tissue. CDDs are very slowly metabolized by the microsomal monooxygenase system to polar metabolites that can undergo conjugation with glucuronic acid and glutathione. They may increase the rate of their own metabolism by inducing both phase I and phase II enzymes. The major routes of excretion of CDDs are the bile and the faeces, though smaller amounts are excreted in the urine and via lactation. (L177)

来源:Toxin and Toxin Target Database (T3DB)

毒理性

• 毒性总结

CDDs通过结合芳基烃受体并随后改变某些基因的转录来产生其毒性作用。对Ah受体的亲和力取决于特定CDD的结构。基因表达的改变可能是由于Ah受体及其异二聚体形成伙伴芳基烃受体核移位子与基因调控元件的直接相互作用，或者启动磷酸化/去磷酸化级联反应，随后激活其他转录因子。受影响的基因包括几个癌基因、生长因子、受体、激素和药物代谢酶。这些基因的转录/翻译改变被认为是CDDs大多数毒性作用的原因。

CDDs cause their toxic effects by binding to the aryl hydrocarbon receptor and subsequently altering the transcription of certain genes. The affinity for the Ah receptor depends on the structure of the specific CDD. The change in gene expression may result from the direct interaction of the Ah receptor and its heterodimer-forming partner, the aryl hydrocarbon receptor nuclear translocator, with gene regulatory elements or the initiation of a phosphorylation/dephosphorylation cascade that subsequently activates other transcription factors. The affected genes include several oncogenes, growth factors, receptors, hormones, and drug-metabolizing enzymes. The change in transcription/translation of these genes is believed to be the cause of most of the toxic effects of CDDs. (L177)

来源:Toxin and Toxin Target Database (T3DB)

毒理性

• 致癌性证据

评估：有有限的实验动物证据表明，1,2,3,6,7,8-和1,2,3,7,8,9-六氯二苯并-对-二噁烷混合物的致癌性。其他多氯代二苯并-对-二噁烷对人体致癌性无法分类（第3组）。/多氯代二苯并-对-二噁烷/

Evaluation: There is limited evidence in experimental animals for the carcinogenicity of a mixture of 1,2,3,6,7,8- and 1,2,3,7,8,9-hexachlorodibenzo-para-dioxins. Other polychlorinated dibenzo-para-dioxins are not classifiable as to their carcinogenicity to humans (Group 3). /Polychlorinated dibenzo-para-dioxins/

来源:Hazardous Substances Data Bank (HSDB)

毒理性

• 致癌物分类

3, 其对人类致癌性无法分类。

3, not classifiable as to its carcinogenicity to humans. (L135)

来源:Toxin and Toxin Target Database (T3DB)

毒理性

• 健康影响

接触大量氯丹类化合物（CDDs）会导致氯痤疮，这是一种严重的皮肤疾病，其特点是在面部和上半身出现类似粉刺的损伤。CDDs还可能引起肝脏损伤，并导致长期的葡萄糖代谢改变和激素水平的微妙变化。此外，研究还表明CDDs可能会干扰内分泌系统，削弱免疫系统，以及造成生殖损害和出生缺陷、中枢和周围神经系统的病理变化、甲状腺疾病、子宫内膜异位症和糖尿病。(L177, L178)

Exposure to large amounts of CDDs causes chloracne, a severe skin disease with acne-like lesions that occur mainly on the face and upper body. CDDs may also cause liver damage and induce long-term alterations in glucose metabolism and subtle changes in hormonal levels. In addition, studies have shown that CDDs may disrupt the endocrine system and weaken the immune system, as well as cause reproductive damage and birth defects, central and peripheral nervous system pathology, thyroid disorders, endometriosis, and diabetes. (L177, L178)

来源:Toxin and Toxin Target Database (T3DB)

毒理性

• 暴露途径

口服（L177）；吸入（L177）；皮肤（L177）

Oral (L177) ; inhalation(L177) ; dermal (L177)

来源:Toxin and Toxin Target Database (T3DB)

吸收、分配和排泄

在1980年至1982年期间，荷兰收集了19只鸬鹚（Phalacrocorax carbo）、3只苍鹭（Ardea cinerea）和1只大凤头鸊鷉（Podiceps crisatus）。分析了这些食鱼鸟的肝脏中的多氯联苯-p-二噁烷和二苯并呋喃。只有在肝脏中发现了具有2,3,7,8-氯取代模式的同类物。主要成分是2,3,4,7,8-五氯二苯并呋喃和1,2,3,6,7,8-六氯二苯-p-二噁烷，2,3,7,8-四氯二苯-p-二噁烷和1,2,3,7,8-五氯二苯-p-二噁烷也存在。六个混合样本的鳗鱼Anquilla anquilla显示出与这些鸟类中发现的同类化学物质相同的模式。在鳗鱼中，2,3,4,7,8-五氯二苯并呋喃和1,2,3,6,7,8-六氯二苯-p-二噁烷通常在1-5 ng/kg的范围内。由于鳗鱼是鸬鹚的主要食物，这表明这两种同类物在鸬鹚肝脏中具有很强的生物累积性。

In the period 1980 to 1982, 19 cormorants (Phalacrocorax carbo), 3 herons (Ardea cinerea), and 1 great crested grebe (Podiceps crisatus) were collected in The Netherlands. The livers of these fish-eating birds were analyzed for polychlorinated dibenzo-p-dioxins and dibenzofurans. Only congeners with a 2,3,7,8-chlorine substitution pattern were found in the livers. The major component was 2,3,4,7,8-pentachlorodibenzofuran and 1,2,3,6,7,8-hexachlorodibenzo-p-dioxin, 2,3,7,8-tetrachlorodibenzo-p-dioxin, 1,2,3,7,8-pentachloridibenzo-p-dioxin were also present. Six pooled samples of the eel Anquilla anquilla showed the same congeneric pattern of chemicals as found in these bird species. In the eel, 2,3,4,7,8-pentachlorobenzofuran and 1,2,3,6,7,8-hexachlorodibenzo-p-dioxin were generally present in the 1-5 ng/kg range. Since the eel is the cormorant's major food, this indicates strong bioaccumulation for both congeners in the liver of the cormorant.

来源:Hazardous Substances Data Bank (HSDB)

吸收、分配和排泄

多氯二苯并-p-二噁英和多氯二苯并呋喃主要储存在脂肪中，但它们也会通过乳汁排出并被胎盘传递。它们还以较低的浓度出现在血液和主要器官中。/多氯二苯并-p-二噁英/

The polychlorinated dibenzo-p-dioxins and the polychlorinated dibenzofurans are predominantly stored in fat, but they are also excreted in milk and pass the placenta. They also appear in the blood and vital organs at lower concentrations. /Polychlorinated dibenzo-p-dioxins/

来源:Hazardous Substances Data Bank (HSDB)

吸收、分配和排泄

从加拿大五大湖流域的5个市镇在尸检中获取的人类脂肪组织样本中，使用气相色谱-高分辨率质谱法分析了多氯代二苯并二噁烷和多氯代二苯并呋喃。每个市镇的男性和女性供体平均同系物值与之前报告的数据相当。在男性和女性之间以及市镇之间未检测到显著的同系物水平差异。观察到几种同系物及其总同系物浓度（以2,3,7,8-四氯代二苯并二噁烷毒性当量表示）与年龄之间存在正相关关系。

Human adipose tissue samples obtained during autopsies in 5 Canadian municipalities within the Great Lakes basin were analyzed for polychlorinated dibenzodioxins and dibenzofurans using GC-high resolution-MS. The mean congener values for male and female donors in each municipality are comparable with previously reported data. No significant differences in congener levels between male and female and between municipalities were detected. A positive correlation between congener level and age was observed for several congeners as well as for the total congener concn expressed as its 2,3,7,8-tetrachlorodibenzodioxin toxic equivalent.

来源:Hazardous Substances Data Bank (HSDB)

吸收、分配和排泄

对11名死于癌症的患者进行了2,3,7,8-氯代二苯并-p-二恶英的组织分布研究。每个器官和组织中八氯二苯并-p-二恶英的浓度最高，其次是七氯二苯并-p-二恶英，其水平仅次于八氯二苯并-p-二恶英。脾中1,2,3,7,8-五氯二苯并-p-二恶英和1,2,3,6,7,8-六氯二苯并-p-二恶英的水平最高。2,3,7,8-四氯二苯并-p-二恶英也被检测到，且其在性腺中的浓度最高（0.8-3.2 pg/g范围）。从2,3,7,8-四氯二苯并二恶英的毒性当量计算中，最高的当量值来自于一名54岁的死于癌性甲状腺肿的女性。在这11名患者中，这位女性的2,3,7,8-取代的五氯和六氯二苯并-p-二恶英浓度最高。

The tissue distribution of 2,3,7,8-chlorine substituted dibenzo-p-dioxins was conducted in 11 patients who died of cancer. The concn of octachlorodibenzo-p-dioxin was the highest in each organ and tissue and heptachlorodibenzo-p-dioxin was also found at relatively high levels, second only to octachlorodibenzo-p-dioxin. The levels of 1,2,3,7,8-pentachlorodibenzo-p-dioxin and 1,2,3,6,7,8-hexachlorodibenzo-p-dioxin in the spleen were the highest, respectively. 2,3,7,8-Tetrachlorodibenzo-p-dioxin was also detected and its concentration was the highest in the gonad (0.8-3.2 pg/g-range). From the 2,3,7,8-tetrachlorodibenzodioxin toxic equivalent calculations, the highest equivalent value was obtained from a 54 yr old female who died of cancerous goiter. This individual had the highest concentration of 2,3,7,8-substituted penta- and hexachlorodibenzo-p-dioxins among the 11 patients.

来源:Hazardous Substances Data Bank (HSDB)

安全信息

• 危险等级：
  6.1(a)
• 包装等级：
  I
• 危险类别：
  6.1(a)
• 危险品运输编号：
  UN 2811

反应信息

• 作为产物：
  描述：

  alkaline earth salt of/the/ methylsulfuric acid 在 iron(II,III) oxide 、 iron(III) oxide iron hydroxide oxide 、 limonite 、 sodium chloride 作用下， 生成 1,2,3,6,7,8-六氯二苯并-对-二恶英
  参考文献：
  名称：

  在铁矿石烧结中形成二恶英。

  摘要：

  铁矿石烧结是“二恶英”，多氯二苯并对二恶英和二苯并呋喃（PCDD / Fs）的重要来源。本文报道了尝试确定造成PCDD / F形成的材料，条件和机理的尝试（i）通过研究矿石的显着特性（即关于模型有机物的氧化，冷凝和氯化），以及（ii）使用现实生活的材料在微观尺度上模拟工业过程。采用实验设计原理（DOE）。铁矿石的反应性差异很大。褐铁矿/针铁矿“软”矿石是一种非常活泼的氧化催化剂（例如，用于苯和苯酚），该特性可用于清除粗烧结工艺废气，而赤铁矿/磁铁矿“硬”矿石则不是。后者，但是强烈促进苯酚缩合为二苯并呋喃。新建的实验室级微型烧结设备可以部分或整体令人满意地模仿大规模过程。用实际的进料混合物获得的结果表明，在烧结床中二恶英的形成水平足以说明在现实生活中观察到的大部分产出。通过添加约8 ppm（wt）的氯化物作为NaCl，PCDD / F的产量增加了一倍，但是在氯的施用比例与C2Cl4相同的情况

  DOI：

  10.1021/es026292g

文献信息

• PCDD/DF formations by the heterogeneous thermal reactions of phenols and their TiO2 photocatalytic degradation by batch-recycle system
  作者：Hajime Muto、Koki Saitoh、Hitoshi Funayama

  DOI：10.1016/s0045-6535(00)00552-x

  日期：2001.10

  and dibenzofurans (PCDD/DFs) formation by the thermal reactions of phenols with CuCl2 under oxygen flux were carried out in relation to their formation mechanisms. To evaluate the effect of photocatalytic degradation of titanium dioxide (TiO2) thin film prepared by the sol-gel method, the photocatalysis of PCDD/DFs in acetonitrile/water solution by batch-recycle system was conducted. For the thermal reaction

  就酚类与CuCl2在氧通量下的热反应，进行了多氯代二苯并二恶英和二苯并呋喃（PCDD / DFs）的形成机理的研究。为了评价溶胶-凝胶法制备的二氧化钛（TiO2）薄膜的光催化降解效果，采用间歇循环系统对PCDD / DFs在乙腈/水溶液中的光催化性能进行了研究。对于2,4,5-三氯苯酚（2,4,5-TCP）和CuCl2的粉末混合物的热反应系统，总PCDD /的形成速率为8.1 microg / g-2,4,5-TCP / min PCDDs的DFs和6.9 microg / g-2,4,5-TCP / min，与苯酚蒸气/氧气/ CuCl2粉末系统相比，PCDD / DF的总速率高出约40倍。对于2,4,5-TCP的系统，PCDDs主要是通过邻苯氧酚（POP）中间体通过2,4,5-三氯苯酚的缩合反应形成的。对于PCDD / DF光催化降解，大多数PCDD同系物会快速分解，并且在辐照后24小时使用由2
• Emissions of polychlorinated dibenzo-p-dioxins and dibenzofurans from catalytic and thermal oxidizers burning dilute chlorinated vapors
  作者：John R. Hart

  DOI：10.1016/j.chemosphere.2003.10.017

  日期：2004.3

  (ng/dscm)=8.4 exp(-0.0084T degrees C); (2) dioxin/furan production occurs at the combustion catalyst; (3) small variations in temperature cause large changes in the congener distribution of the dioxin and furan isomers; (4) molar TEQ yields from the parent compounds fed to the oxidizers are very small (10(-9)-10(-13)); (5) catalytic and thermal oxidizers may destroy dioxins fed from the ambient air; and (6)

  通过对低（几至几百）百万分之一的氯化和非氯化挥发性有机化合物（VOC）的氧化进行的57次现场测试，发现了多氯二苯并对二恶英和二苯并呋喃（二恶英）的排放。在使用铂，铂/钯或铬（IV）氧化物燃烧催化剂的催化氧化剂中，或在热氧化剂（无催化剂）中发生氧化。催化剂入口温度为293至573摄氏度。热氧化剂的运行温度（火焰后）为773至927摄氏度。报告了有毒的二恶英和呋喃异构体的数据，并进行了加权和表示为国际有毒当量（ 2,3,7,8-四氯二苯并-对-二恶英的TEQ）。最大烟囱排放为1.07 ng / m3 TEQ，发生在293摄氏度。该现场研究的主要结果是：（1）堆中的TEQ水平随工作温度的降低而呈指数增长，经验公式为TEQ（ng / dscm）= 8.4 exp（-0.0084T摄氏度）; （2）在燃烧催化剂处产生二恶英/呋喃；（3）温度的微小变化会导致二恶英和呋喃异构体的同类物分布发生较大变化；
• Emission Factors and Importance of PCDD/Fs, PCBs, PCNs, PAHs and PM₁₀ from the Domestic Burning of Coal and Wood in the U.K.
  作者：Robert G. M. Lee、Peter Coleman、Joanne L. Jones、Kevin C. Jones、Rainer Lohmann

  DOI：10.1021/es048745i

  日期：2005.3.1

  fuels. However, their combined emissions from the domestic burning of coal and wood would contribute only a few percent to annual U.K. emission estimates. Emissions of PAHs and PM10 were major contributors to U.K. national emission inventories. Major emissions were found from the domestic burning for Cl1,2,3DFs, while the contribution of PCDD/F-sigmaTEQ to total U.K. emissions was minor.

  本文介绍了当煤和木材经过受控燃烧实验时针对一系列持久性有机污染物（POPs）得出的排放因子（EFs），旨在模拟空间供暖的家庭燃烧。排放了各种各样的持久性有机污染物，煤炭的排放量高于木材的排放量。对于颗粒物，PM10（大约10 g / kg燃料）和多环芳烃（对于sigmaPAHs大约100 mg / kg燃料）获得了最高的EF。对于氯化物，多氯联苯（PCB）的EF最高，而多氯萘（PCN），二苯并-对-二恶英（PCDD）和二苯并呋喃（PCDF）的丰度较低。对于sigmaPCB，EF大约为1000 ng / kg燃料，对于sigmaPCNs大约为100s ng / kg燃料，对于sigmaPCDD / Fs大约为100 ng / kg燃料。该研究证实，一氯化至三氯化二苯并呋喃Cl1,2,3DFs是低温燃烧过程（如煤炭和木材的国内燃烧）的有力指标。结论是，在固体燃料燃烧期间通常形成许多PCB和PC
• Polychlorinated Dibenzo-<i>p</i>-dioxin/Polychlorinated Dibenzofuran Releases into the Atmosphere from the Use of Secondary Fuels in Cement Kilns during Clinker Formation
  作者：Esteban Abad、Karell MartÍnez、Josep Caixach、Josep Rivera

  DOI：10.1021/es049641a

  日期：2004.9.1

  this study was to evaluate the influence of using waste materials, such as tires or meat meal, as a secondary fuel during clinker production on the polychlorinated dibenzo-p-dioxin (PCDD)/polychlorinated dibenzofuran (PCDF) emission levels to the atmosphere. For this purpose, three different cement plants in Spain were chosen to conduct the project in different sampling episodes. Different materials were

  这项研究的目的是评估在熟料生产过程中使用废料（例如轮胎或肉粉）作为二次燃料对多氯二苯并对二恶英（PCDD）/多氯二苯并呋喃（PCDF）排放水平的影响。大气层。为此，在西班牙选择了三个不同的水泥厂以不同的采样次数进行该项目。每家工厂分别评估了不同的材料：第一家工厂在窑中添加了肉粉，第二家工厂使用了废旧轮胎，第三家工厂使用了两者的混合物。在所有情况下，PCDD / F排放值均保持在欧盟指令规定的0.1 ng I-TEQ / Nm3的限值以下，范围在0.001至0.042 ng I-TEQ / Nm3的范围内。在大多数情况下，对总TEQ的主要贡献来自2,3,7，8-四氯二苯并呋喃的含量较高，而2,3,4,7,8-五氯二苯并呋喃的含量较高，因为其TEF为0.5。其余15种有毒同源物仅对TEQ贡献很小。此外，与使用常规燃料从西班牙水泥窑厂获得的报告数据相比，没有发现显着差异。这一事实表明，添加废轮胎或肉粉对PCDD
• Catalytic NOx reduction with simultaneous dioxin and furan oxidation
  作者：Marcel Goemans、Patrick Clarysse、Joseph Joannès、Petra De Clercq、Silvia Lenaerts、Karel Matthys、Kris Boels

  DOI：10.1016/s0045-6535(03)00255-8

  日期：2004.3

  of a Flemish incineration plant is presented, covering the history, the design procedure of the catalyst, relevant process data and the financial aspects. A reliable PCDD/F-destruction by means of oxidation by the catalyst to typical values of 0.001 ng TEQ/Nm3 has been demonstrated. At the same time, NOx- and CO-emissions are reduced by 90% and 20% to about 50 mg/Nm3 and below 10 mg/Nm3, respectively

  讨论了市政垃圾焚烧炉低粉尘区催化烟气净化组件的工程，构造，性能和运行成本。为此，介绍了佛拉芒焚化厂的案例研究，涵盖了历史，催化剂的设计程序，相关的工艺数据和财务方面。通过催化剂氧化达到典型值0.001 ng TEQ / Nm3的可靠PCDD / F破坏已得到证明。同时，NOx和CO的排放量分别减少了90％和20％，分别为50 mg / Nm3和10 mg / Nm3以下。

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